Main Article Content
Abstract
Hepatitis B infection (HBV) is known as the primary driver of hepatitis B. Such an infection can assault and damage the entire of the liver cells. The huge issue with the hepatitis B infection is the vast majority with its contamination have no manifestations, particularly when they are either as of late tainted or their contaminated is constant. In the current study, the level of some boundaries (GPT, GOT, ALP, bilirubin and CRP) were estimating for patients with constant hepatitis B. Sixty examples were taken in the investigation with age of 27.35±7.61year. In chronic patients, the levels of GPT, GOT, ALP, bilirubin and CRP were 25, 30, 30 and 30% with significant different with those of controls (15, 10, 10 and 10%). There was critical contrast in GPT and GOT, but as no high distinction in bilirubin and CRP. The current examination suggests that a need to check a wide range of viral hepatitis B to ensure the contamination stage, particularly during medical procedures and pregnancy.
Keywords
Article Details
The authors transfer the copyrights of their papers to the Iranian Society of Ichthyology. However, the information could be used in accordance with the Creative Commons licence (Attribution-NonCommercial 4.0 International (CC BY-NC 4.0)
References
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Do, A. & Reau, N.S. 2020. Chronic viral hepatitis: current management and future directions. Hepatology Communications 4(3): 329-341.
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Loomba, R. & Liang, T.J. 2007. Treatment of chronic hepatitis B. Antiviral Therapy (Suppl 3): H33-H41.
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McMahon, B.J. 2009. The natural history of chronic hepaatitis B virus infection. Hepatology 49(Suppl): S45–S55.
Ribeiro, A.J.; Yang, X.; Patel, V.; Madabushi, R. & Strauss, D.G. 2019. Liver microphysiological systems for predicting and evaluating drug effects. Clinical Pharmacology & Therapeutics 106(1): 139-147.
Souza Pires-Neto, O.; Silva Graça Amoras, E.; Queiroz, MAF.; Demachki, S.; Silva Conde, S.R.; Ishak, R.; Cayres-Vallinoto, I.M.V. & Vallinoto, A.C.R. 2020. Hepatic TLR4, MBL and CRP gene expression levels are associated with chronic hepatitis C. Infect. Genetics and Evolution 80: 104200.
World Health Organization, 2020. "Clinical Chemistry Analyzer" (PDF). (Accessed 15 May 2020).
Zaidan, T.A. & Saod, W.M. 2016. Renal function status of Fallujah patients (Iraq) infected with chronic hepatitis B. Journal of University of Babylon 24(7).
References
Cheng, Y.; Dubovoy, N.; Hayes-Rogers, M.E.; Stewart, J. & Shah, D. 1999. Detection of IgM to hepatitis B core antigen in a reductant containing, chemiluminescence assay. Journal of Immunological Methods 230(1-2): 29-35.
Do, A. & Reau, N.S. 2020. Chronic viral hepatitis: current management and future directions. Hepatology Communications 4(3): 329-341.
Ganem, D. & Schneider, R.J. 1996. Hepadnaviridae and Their Replication. Fields Virology, 4.
Jungen, M.J.; Ter Meulen, B.C.; Van Osch, T.; Weinstein, H.C. & Ostelo, R. 2019. Inflammatory biomarkers in patients with sciatica: a systematic review. BMC Musculoskelet Disord 20(1): 1-9.
Kahila Bar‐Gal, G.; Kim, M.J.; Klein, A.; Shin, D.H.; Oh, C.S.; Kim, J.W. & Shouval, D. 2012. Tracing hepatitis B virus to the 16th century in a Korean mummy. Hepatology 56(5): 1671-1680.
Keeffe, E.B.; Dieterich, D.T.; Han, S.H.; Jacobson, R.M.; Martin, P. & Schiff, E.R. 2006. A treatment algorithm for the management of chronic hepatitis B virus infection in the United States: an update. Clinical Gastroenterology and Hepatology 4: 936-62.
Kuhns, M.C. & Busch, M.P. 2006. New strategies for blood donor screening for hepatitis B virus: nucleic acid testing versus immunoassay methods. Molecular Diagnosis & Therapy 10: 77–91.
Lai, M.; Hyatt, B.J.; Nasser, I.; Curry, M. & Afdhal, N.H. 2007. The clinical significance of persistently normal GPTin chronic hepatitis B infection. Journal of hepatology 47(6): 760-7.
Lee, Y.; McKechnie, T.; Doumouras, A.G.; Handler, C.; Eskicioglu, C.; Gmora, S.; Anvari, M. & Hong, D. 2019. Diagnostic Value of C - reactive protein Levels in Postoperative Infectious Complications after Bariatric Surgery: a Systematic Review and Meta-Analysis. Obesity Surgery 29(7): 2022-2029.
Loomba, R. & Liang, T.J. 2007. Treatment of chronic hepatitis B. Antiviral Therapy (Suppl 3): H33-H41.
Loomba, R.; Rowley, A.; Wesley, R.; Liang, T.J.; Hoofnagle, J.H. & Pucino, F. 2008. Systematic review: the effect of preventive lamivudine on hepatitis B reactivation during chemotherapy. Annals of Internal Medicine 148: 519-528.
McMahon, B.J. 2009. The natural history of chronic hepaatitis B virus infection. Hepatology 49(Suppl): S45–S55.
Ribeiro, A.J.; Yang, X.; Patel, V.; Madabushi, R. & Strauss, D.G. 2019. Liver microphysiological systems for predicting and evaluating drug effects. Clinical Pharmacology & Therapeutics 106(1): 139-147.
Souza Pires-Neto, O.; Silva Graça Amoras, E.; Queiroz, MAF.; Demachki, S.; Silva Conde, S.R.; Ishak, R.; Cayres-Vallinoto, I.M.V. & Vallinoto, A.C.R. 2020. Hepatic TLR4, MBL and CRP gene expression levels are associated with chronic hepatitis C. Infect. Genetics and Evolution 80: 104200.
World Health Organization, 2020. "Clinical Chemistry Analyzer" (PDF). (Accessed 15 May 2020).
Zaidan, T.A. & Saod, W.M. 2016. Renal function status of Fallujah patients (Iraq) infected with chronic hepatitis B. Journal of University of Babylon 24(7).